I found this little study and thought it was interesting in that maybe this would help with brain fog. We talk about daily meditation in our fibromyalgia ebook. There are many fibromyalgia symptoms that meditation can help with. Let us know if you have tried meditation and your results at info@ridfibromyalgia.com.
Improve Your Concentration
Researchers have found that even short periods of daily meditation can improve both focus and performance.
Subjects in a study were split into two categories. The first, consisting of those new to meditation, underwent an eight-week course including 30 minutes of daily meditation.
The second group, which was composed of those already experienced with meditation, attended a full-time, one month retreat.
Even for those completely new to the practice, meditation improved performance and the ability to focus attention. Their scores on tests measuring cognitive function improved after only a few weeks.
Throughout, participants took computer tests that measured response speeds and accuracy. At the beginning of the study, those who were experienced in meditation demonstrated better voluntary focus, task management, and goal setting skills.
But after completing the eight-week course, those who were new to meditation showed greater improvement in focusing their attention.University of Pennsylvania May 9, 2007Science Daily June 26, 2007
Monday, July 16, 2007
Tuesday, July 10, 2007
Disability, Hope or No Hope
Found this comment about fibromyalgia and disability. Have you had any luck with getting disability? Let me know of your comments.
"At the moment, there is no entry in Social Security Disability 's Listing of Impairments manual for the condition known as Fibromyalgia, also known as fibromyositis and Myofacial Pain Syndrome (The disability listing's, of course, provide the approval criteria for a number of different impairments ranging from amputations to seizure disorder). Nevertheless, many claimants with fibromyalgia apply for disability, and many go on to win their cases. For this reason, the fibromyalgia disability claimant who's been denied should not give up on their case. Instead, they should pursue their disability claim through the appeals process, keeping in mind how the Social Security Administration views this particular impairment."
For more info on how to gain disability, visit www.ridfibromyalgia.com/how.html.
"At the moment, there is no entry in Social Security Disability 's Listing of Impairments manual for the condition known as Fibromyalgia, also known as fibromyositis and Myofacial Pain Syndrome (The disability listing's, of course, provide the approval criteria for a number of different impairments ranging from amputations to seizure disorder). Nevertheless, many claimants with fibromyalgia apply for disability, and many go on to win their cases. For this reason, the fibromyalgia disability claimant who's been denied should not give up on their case. Instead, they should pursue their disability claim through the appeals process, keeping in mind how the Social Security Administration views this particular impairment."
For more info on how to gain disability, visit www.ridfibromyalgia.com/how.html.
Monday, July 9, 2007
Findings offer new theraputic perspective in Fibromyalgia Sufferers
This data was posted on pub med as a study that may have implications for better treatment down the road for fibromyalgia patients. Yeah! Until then, check out http://www.ridfibromyalgia.com/.
All data obtained in experimental animal pain models support the role of nerve growth factor (NGF) as a putative candidate intervening in the pathogenesis of chronic pain, including chronic daily headache (CDH). Few studies have been carried out to establish its role in maintaining pain states in humans. The present study was aimed at investigating cerebrospinal fluid (CSF) levels of NGF and brain-derived neurotrophic factor (BDNF), both measured by sensitive immunoassay, in 20 chronic migraine (CM) patients and 20 patients affected by primary fibromyalgia syndrome (PFMS), compared with those of 20 age-matched control subjects. Significantly higher levels of both neurotrophins and glutamate were found. A significantly positive correlation emerged between CSF values of BDNF and those of NGF (r = .61, P < .001; r = .53, P < .01) and glutamate (r = .44, P < .02; r = .51, P < .01) in CM and PFMS patients, respectively. These findings suggest the possibility of a NGF-mediated up-regulation of BDNF involved in the pathophysiological events underlying long-term neuroplastic changes in persistent chronic painful conditions, such as CM and fibromyalgia. NGF might indirectly exert its effect through enhancing glutamatergic transmission via BDNF. The above mechanisms could account for sustained central sensitization in both chronic pain states. PERSPECTIVE: This article presents findings of higher NGF and BDNF levels correlated to increased glutamate levels in the CSF of both chronic migraine and fibromyalgia patients. This opens new insights into the pathogenic mechanisms of chronic pain and offers clinicians new therapeutic perspectives targeting the above mechanisms in both painful disorders.
All data obtained in experimental animal pain models support the role of nerve growth factor (NGF) as a putative candidate intervening in the pathogenesis of chronic pain, including chronic daily headache (CDH). Few studies have been carried out to establish its role in maintaining pain states in humans. The present study was aimed at investigating cerebrospinal fluid (CSF) levels of NGF and brain-derived neurotrophic factor (BDNF), both measured by sensitive immunoassay, in 20 chronic migraine (CM) patients and 20 patients affected by primary fibromyalgia syndrome (PFMS), compared with those of 20 age-matched control subjects. Significantly higher levels of both neurotrophins and glutamate were found. A significantly positive correlation emerged between CSF values of BDNF and those of NGF (r = .61, P < .001; r = .53, P < .01) and glutamate (r = .44, P < .02; r = .51, P < .01) in CM and PFMS patients, respectively. These findings suggest the possibility of a NGF-mediated up-regulation of BDNF involved in the pathophysiological events underlying long-term neuroplastic changes in persistent chronic painful conditions, such as CM and fibromyalgia. NGF might indirectly exert its effect through enhancing glutamatergic transmission via BDNF. The above mechanisms could account for sustained central sensitization in both chronic pain states. PERSPECTIVE: This article presents findings of higher NGF and BDNF levels correlated to increased glutamate levels in the CSF of both chronic migraine and fibromyalgia patients. This opens new insights into the pathogenic mechanisms of chronic pain and offers clinicians new therapeutic perspectives targeting the above mechanisms in both painful disorders.
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